African AIDS: Vaccine Epidemic - AIDS the Real Story (6)

African AIDS: Vaccine Epidemic

 AIDS: The Real Story – Section 3 (Part 6)

  
 “In the recent WHO smallpox campaign, needles we re-used forty to sixty times. The main method of ‘sterilization’ was waving the needle across a flame…              “WHO [World Health Organisation] information indicates that the AIDS table of Central Africa matches the concentration of smallpox vaccinations, i.e., the greatest spread of HIV infection coincides with the most intense immunization programs. Thus, Zaire… had 36,000,000 people immunized with the smallpox vaccine. Next Zambia, with nineteen million, followed by Tanzania with fifteen million, Uganda with eleven million, Malawi with eight million, Ruanda (Rwanda) with 3.3 million and Burundi with 3.2 million. Brazil, the only South American country covered by the smallpox eradication campaign, had the highest incidence of AIDS in that part of the world.
 
             “The theory – that the AIDS epidemic in Africa may have been triggered by the smallpox eradication program – has sparked intense debate among scientists. You may not have heard about this debate, but an urgent call for evidence to support the idea has been demanded by the World Health Organization. The theory was discussed by WHO officials in Autumn [1987]. No follow-up data are available from the smallpox eradication campaign because no systemic studies of the complications produced by the mass immunization have been done(!)” the late Dr Robert Mendelsohn wrote in 1987.[1]

               WHO figures for March 1988 showed that the majority of African cases were concentrated in the urban areas of the following countries; Zaire, Zambia, Tanzania, Uganda, Rwanda, Burundi, Congo and the Central African Republic. Zambia was previously known as Northern Rhodesia. Tanzania used to be called Tanganyika/Zanzibar; the recent history of Central Africa has altered many population bases. But whether sheer numbers of cases or population densities are considered, those countries on Mendelsohn’s list still rated the highest incidences of AIDS in Africa.

             According to Dr Robert W. Dwyer, director of Project SIDA (the Zairian AIDS research program) in March 1988, “the epidemic now looks like it is beginning to level off… there is no evidence of any increase in several years.” By contrast, AIDS cases apparently increased tenfold between the late 1970s and 1980s.
 
             Dr Ryder’s group found no increase in the infection rate of blood donors or pregnant women in the Zaire capital, Kinshasa, indicating HIV infection had leveled off.[2]
 
             “A distressing example of how HIV is blood-borne comes from a study of children between 1 and 24 months old and infected by the virus but whose mothers are not infected. The babies must, therefore, have become infected after birth. Compared with uninfected children of the same age, the infected children have received an average of 44 medical injections (vaccinations not included) compared with an average of 23 medical injections for uninfected children. The relatively large number of injections that even uninfected, healthy children in Africa has led to a small study of fifty mothers. 84% of these mothers believed that medication by injection was more effective than oral medication. Virtually all the women wanted their children to receive injections rather than pills…
 
             “For many reasons, often financial, health workers may not sterilize or discard needles and syringes after use. This is true especially in small or isolated clinics. The stage is set for injections to help the spread of HIV in Africa.”[3] This was written by Dr Jonathan Mann, Director of the WHO special programme on AIDS in 1987.
 
             Smallpox vaccination in the ‘Third World’ did not begin with the WHO. Whole, ‘live’ virus vaccines were used across Africa from at least the 1920s, when scant medical records began to be kept. In the mid-1920s Rhodesia (later to become Zambia and Zimbabwe) was surrounded by states where “smallpox was rife” and “the vaccination state was not as high… as the [Rhodesian] authorities would have liked it to be.
 
        “Many groups of people were appointed as public vaccinators. Native commissioners, police, compound inspectors, all carried out vaccinations. Certain public vaccinators received a penny per vaccination performed and from this arises the, possibly apocryphal, story that when some of these gentlemen ran out of the vaccine lymph they had recourse to condensed milk in order to avoid losing their penny! In any case, the lack of refrigerated storage and transport for vaccine lymph must have given rise to a great deal of wastage.”[4]
 
             In Rhodesia in 1924; “The only laboratory services existed in Salisbury. The Salisbury Public Health Laboratory being combined with the Pasteur Institute which was still manufacturing a rabies vaccine…”[5] In his 1930 Annual Report the Rhodesian Medical Director draws attention to “The epidemic occurrence of smallpox which has been prevalent in the colony during the last three years and the need for additional preventive measures against the disease.”[6] These quotes were recorded in the Central African Journal of Medicine.
 
             Even if HIV can be isolated in early African blood samples taken prior to the 1970s WHO vaccination program, colonial policemen and ‘compound inspectors’ were probably injecting earlier versions of unknown organisms into humans along with primitive vaccinia back in the 1920s.
 
             The medical services required the vaccine programs to boost their almost non-existent medical facilities. Even to the present day, many of the medical journals and texts quoted here appear to need numerous full-page glossy advertisements from pharmaceutical companies to afford publication.
 
             After the beginning of the spread of AIDS in the West in the late 1980s, the US Public Health Service stated that the heterosexual portion of the infection rate for AIDS in Africa was ninety percent, a figure promulgated to this day. This is easily proven to be incorrect.

              Among African adults in 1987, the highest proportion of HIV-infected people were from 16 to 29 years of age. Children from 1 to 14 years old in Kinshasa, Zaire had the lowest prevalence of only one percent – probably due to infection from the mother. These children were born after 1973. The most affected group was born between 1958 and 1971. Random Samples of infection rates (1987) showed the following figures: Lusaka, Zambia – 17% (128 healthy persons) Kampala, Uganda – 11% (370 blood donors) Bangui, Central African Republic – 4% (1,263 randomly selected people) Cameroon: 1% (1,273 – family surveys) [7] 

              “Current evidence suggests that some people develop AIDS within a year or so of infection, but that four to six years is a more typical incubation period. In some cases the time between catching the virus and developing the disease may be even longer…”[8] ble 2

First 500 Cases of AIDS Diagnosed in Mama Yemo Hospital, Kinshasa, Zaire (1986)

Note the female to male ratios in mortality figures.

Spread of HIV in Africa  

  

The spread of AIDS in West Africa seemed to be less thorough than in Central, Eastern and parts of Southern Africa, and also involved different strains of HIV. [9]
 
             “…because the cost of screening one blood donation in some poor African nations is approximately three times the entire per-capita expenditure for medical expenses, the procedure is virtually never performed and the national blood supply remains thoroughly contaminated. In some areas, the prevalence of HIV in the blood supply is estimated to be 8-10%; since the average transfusion requires several pints of blood, a person receiving a transfusion is practically guaranteed infection.” [10]
 
             “…in (African) areas where 10% of the healthy population is infected, about the same proportion of blood donors are also likely to be infected. The spread of HIV by contaminated blood transfusions is a tremendous problem in Africa, which could be eliminated if the infrastructure and financial means existed to test blood for antibodies to HIV. In Africa today, the risk of blood recipients may be as high as one in ten, yet in many areas blood is still not screened.
 
          “The spread of HIV in Africa obeyed the same basic biological laws which seemed to cause the spread of the virus in Europe and the US…” These statements come from WHO AIDS director Dr Jonathan Mann.[11] 
 
         In Africa roughly half the people with AIDS are female. This is the single major fact that has promoted a description of AIDS as being ‘heterosexually transmitted’ in Africa and elsewhere (yet in the West this sexual balance in infection rates isn’t the case). The rest of the ‘evidence’ for heterosexual non-anal transmission is all circumstantial, consisting of early African interviews with and surveys of small groups of sex workers and their clients.
 
            No verified studies of heterosexual transmission have been reported; where there are ‘studies’ for homosexual and other forms of HIV transmission, there are only ‘models’ for heterosexual spread – and all of these for Africa were statistically flawed from the outset, as they failed to account for local blood banks being “thoroughly contaminated” and the smallpox vaccination program being a (or the)prime transmission vector.
 
         In the West, the ratio of females to males with the syndrome is much lower than in Africa; only about 10% of cases in the US and Europe are women,[12] and these can virtually all be traced to ‘non-heterosexual’ (neither vaginal nor oral sex) vectors, where they can be traced at all. WHO figures continually showed as many ‘unknown vector’ cases as those presumed to be heterosexual – and as demonstrated in Section One of this report, presumption was the only reason many AIDS sufferers were classified as having been infected by ‘heterosexual transmission’.
 
          Are there any signs of HIV infection in Africa dating back to the WHO smallpox eradication program? Blood samples have been taken from African patients and frozen by the WHO and others for decades. Unfortunately, possible contamination with other bodies and frozen storage has meant that tests for HIV in these old samples are inconclusive.
 
         Dr Jonathan Mann, director of the WHO’s AIDS program said that more reliable data showed that a blood sample from Central Africa (Kinshasa, Zaire) seemed to contain HIV antibodies in 1959, and that a Danish surgeon working in Africa from 1972-75 apparently developed AIDS in 1976.
 
          One suspect batch of stored blood samples taken in 1972-73 seems to show that HIV antibodies were present in the blood of fifty Ugandan children out of a group of 75.

          Researching medical records one finds that that suddenly, in about 1978, an epidemic illness related to AIDS began in Africa. One Belgian researcher and his colleagues found there was a sudden epidemic of cryptococcal meningitis (a disease co-factor of AIDS) in 1978, that paralleled the African spread of HIV. “Slim disease” apparently began in Uganda in the early 1980s; an epidemic of Kaposi’s sarcoma was noted in Zambia in 1982, and in Rwanda in 1983 doctors found a marked increase in candidal oesophagitis, a fungal infection commonly found in the digestive tracts of AIDS sufferers. At this time there was no test for HIV.[13] 

           If an epidemic of illnesses related to AIDS began in Africa around 1978 and the normal onset time of the syndrome was four to six years, then this would place the beginning of the widespread advent of HIV in Africa at around 1972-74. African children born after 1973 are less affected than those who came before.
 
         Some researchers, including Dr Douglass (cited earlier), have researched the smallpox vaccination programs conducted in Africa at that time. Strecker, Mendelsohn, Pearce Wright, Douglass, Rifkin and others claim that the epidemiology of AIDS corresponds precisely with the WHO smallpox vaccination program. Douglass goes so far as to say that a particular vaccination program (referred to in a 1972 WHO report of a 1970 NIH conference) was laced with HIV.[14]
 
         This is the research he refers to; “In relation to the immune response, a number of useful experimental approaches can be visualized. One would be a study of the relationship of HL-A type to the immune response, both humeral and cellular, to well-defined bacterial and antiviral antigens during preventative vaccination. This approach would be particularly informative when applied to sibships.”[15]
 
         ‘Sibships’, or brother and sister relations, would be used to determine changes in the effects of various vaccines within similar environments and genetic patterns (The 1972 WHO Bulletin cited in Section Two of this report raised similar questions, regarding the proposal to see if disease organisms could be rendered more virulent by a ‘hypothetical’ novel agent – an organism whose description matches HIV).
 
         This researcher has been unable to find data that confirms or refutes assertions by Douglass that vaccination programs subsequent to this July 1970 NIH workshop was deliberately contaminated with HIV; it’s just as possible that HIV had evolved into its present (human affecting) form from related animal retroviruses contaminating vaccine cultures around this time, given the normal cross-species infection practices and prevalence of accidents in biological research labs.
 
         But assertions that HIV was created in Fort Detrick/NIH/NCI have been made repeatedly over the past decades and this possibility must continue to be considered until proven incorrect. As demonstrated in earlier sections of this report, circumstantial evidence and expert opinions in support of this position do exist. It has often been stated that it would be very difficult, if not impossible, for HIV to evolve of its own accord (DR R.J. Biggar, Lancet, and others – see earlier sections).
 
         “HIV may have arisen by mutating and making the jump from other primates to humans, following a route broadly analogous to that whereby smallpox, measles and other viruses probably entered the human population from domestic animals some 10,000 years ago…”, according to some.[16] But this opinion is refuted by many reputable virologists, as we have seen.
 
         The two most likely origins of HIV (and causes of the sudden widespread advent of AIDS) are; by accident due to the repeated admixture of continually evolving retroviruses into millions of simian and human tissue cultures and bloodstreams - or by combination in a biological research laboratory and subsequent dissemination (either by accident or deliberately) to groups of people. Multinational bulk blood purchasing spread HIV widely around the world in either case.    

African Warnings  

The major WHO smallpox vaccination program began in 1967; it may be that anomalous signs of immuno-deficiency were not recognised in the early years of the program.
 
             AIDS in the West was first diagnosed in young and middle-aged homosexual men, many of whom were affected by an uncommon form of lymphatic cancer known as Kaposi’s sarcoma (KS). This form of cancer is normally found only in older men, which was one of the major indications in the West that something unusual was happening with younger gay males and others.
 
             Yet a study in The Central African Journal of Medicine, in January 1973 reports anomalous KS among younger black Rhodesian Africans.[17]
 
             “Fifty-eight new cases presenting at Harare Hospital (Salisbury) between 1967 and 1971 were reviewed…
 
             “Table 1 shows the spread of occurrence of the tumour throughout all age groups but the maximum incidence is at 60 years of age and over. It is of interest that ten cases occurred between the ages of 20 and 29.” (See Table 1). 

                                                  Table 1: Ages of cases seen
 
Ages 1 year to 9 years                          :           3 cases                       
Ages 10 years to 19 years                   :           2 cases                       
Ages 20 years to 29 years                   :           10 cases                       
Ages 30 years to 39 years                   :           7 cases                       
Ages 40 years to 49 years                   :           8 cases                       
Ages 50 years to 59 years                   :           4 cases                       

Ages 60 and over                                     :           24 cases

“Only three patients were female and the age range was 13 years, 25 years and 65 years.” 

              Two cases “had a history of pulmonary tuberculosis and were receiving sanatorium treatment when they developed KS…
 
             “Three cases complained of cough and shortness of breath with chest pain…”[18]
 
             The WHO smallpox eradication program began its immunisation campaign in these areas in 1967 (see Section 2). It’s easy to see that an anomalous incidence of KS in young men could draw widespread attention in the West in 1981. But similar findings in Rhodesia in 1973 were likely to be ignored. A little further on, however, we find that attempts were made in Rhodesia to treat KS by applying vaccinia directly to the lesions:
 
           “There has been a great deal of discussion on the association of KS with other tumours and with other diseases (O’Brien & Brasfield, 1967). It is patently evident to us in this country that there is an association with tuberculosis…” By the 1980s this was common with HIV infection.

 
             “In view of the fact that there may be a certain association with immunity, inoculation of some of the tumours with vaccinia virus has produced regression of the surface portion of the Kaposi’s nodule but the tumour in deep tissues has remained unchanged.”[19] The previous year’s Journal describes how attempts to investigate “The therapeutic value of vaccinia virus in malignant melanoma using direct, intradermal inoculation, intravenous injection and injection of regional lymph metastases has already been reported… However, there has been little success with direct lymph node inoculation…”
 
             The authors find no “clinical, histological or virological evidence to support the use of vaccinia virus” in treating lymphatic cancers. “However, in our service, direct inoculation of metastases with vaccinia virus has become the treatment of choice.”[20]
 
              An earlier edition of the same Journal reports on carcinomas in smallpox vaccination scars.[21] In their conclusion the authors outline some problems with smallpox vaccination in the field;
 
             “Amongst the complications of vaccination we appreciate that infections can occur at the vaccination site, non-specific eruptions can appear and generalized vaccinia and progressive vaccinia can develop… Encephalitis is not infrequent.”
 
             Through the early 1970s (at the very least), experiments with vaccinia virus were conducted on black African patients in Rhodesia. In many cases the vaccinia (along with contaminating organisms) was pumped directly into the lymphatic systems or skin tumours of patients with melanomas (lymphatic cancers) – including Kaposi’s sarcoma, a common co-factor of AIDS in the early years of its appearance. Again there are documented “complications” associated with vaccinia virus that no-one cared to investigate.
 
             We are told that ‘African AIDS’ is easily transmitted ‘heterosexually’ while its Western counterpart is not, despite the fact that these are the same organisms infecting the same species. It’s obvious that this argument is flawed. There is no proof of non-anal heterosexual transmission in Africa; just as is the case in the West, this assumption was based on inferences gained from statistical research (often using small numbers of individuals in unrepresentative groups, such as sex workers in single cities) and voluntary interviews. The first section of this report has demonstrated that similar Western figures are misrepresentative even when they are accurate.
 
           The most likely explanation for the equal distribution of AIDS between the sexes in Africa is that most were infected in the World Health Organisation vaccination programs. HIV was further spread primarily by contaminated blood banks and blood products and intravenous drug use.
 
             Corroboration comes from the fact that HIV is not transmitted easily (if at all) from females to males or from receptive partners in anal sex to the active partner. 

           In this researcher’s opinion, the misidentification of the true source of AIDS in Africa and other parts of the ‘third world’ – the WHO smallpox eradication campaign – is one major reason the world was told that AIDS is a heterosexually (vaginally) transmitted disease when it obviously is not (Pointing out this obvious fact – when hardly anyone uses condoms in practice during intercourse – is like pointing out that the emperor has no clothes. Most people – including well-paid researchers and scientists - seem to overlook the obvious fact that HIV is NOT spread by vaginal intercourse and act as if they believe the opposite were the case – except when making love!).  This misclassification succeeded in drawing attention away from the likely origin of HIV/AIDS itself – the contamination of (smallpox) vaccinia with primate, sheep and cow organisms, the identities of which may already be known (see Section Two – The Virus Engineers).
 
            That this hypothesis should be further investigated with determination by the World Health Organisation and others should be obvious to any who have read this far. An accurate identification and description of the disease organism may depend upon such investigations.

        According to a report by the Panos Research Institute in conjunction with the Norwegian Red Cross called AIDS in the Third World, issued in November 1986, “the possibility that these immunisation drives could be helping spread AIDS and diseases like polio is an agonizing one for many organisations like WHO and UNICEF, and for thousands of doctors.” The report predicted that at least one million Africans could die of AIDS in the following ten years, mainly in Central Africa.[22]  -           

Continues…

          -          by R. Ayana-            

-          See

AIDS the Real Story - Prologue

AIDS the Real Story Section 1 – The Heterosexual Myth

Section 1 Part 2 - Very Hard to Catch

-          Se AIDS the Real Story Section 2 Intro - Smallpox Vaccines
AIDS the Real Story Section 2 – The Virus Engineers

 See Section 2 Part 2 - Bacteriological Warfare

Section 2 Part 4 - Mistake Waiting to Happen 

Part 5 - 'Gay' AIDS - Early Warning

AIDS the Real Story Section 3 - African AIDS

For more truths about AIDS see http://nexusilluminati.blogspot.com/search/label/aids

images - http://southafrica-for-dummies.com/image-files/aids-in-southafrica-stats.jpg
http://www.globalenvision.org/articleimages/_image/chart_educatormortality_pLoS.jpg

http://www.codezonline.com/content_images/smallpox4-thumb.jpg
http://news.bbc.co.uk/olmedia/1780000/images/_1781032_aidsvictim_ap150.jpg

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[1]  Dr Robert S, Mendelsohn, Australasian Health and Healing, Vol 7 No 2, December 1987

[2]  Washington Post, 21-3-1988, Sydney Morning Herald 23-3-88

[3]  Dr Jonathan Mann, New Scientist 26-3-87

[4]  A Review of the Development of the Health Services of Rhodesia from 1923 to the Present Day; Central African Journal of Medicine, Vol 18 No 12, December 1972, p 244

[5]  Ibid p. 247

[6]  Ibid Vol 19 No 1 January 1973

[7]  Communicable Diseases Intelligence (CDI) 87/7 from WER No 11, 13-3-87

[8]  Roy M. Anderson, FRS & Robert M. May, FRS, New Scientist 26-3-87 p 56

[9]  Ibid, Dr Jonathan Mann

[10]  Michael Fumento, The Bulletin (Australia) 22-3-88

[11]  Dr Jonathan Mann, New Scientist 26-3-87

[12]  Figures; Dr Jonathan Mann, Ibid, & CDI 88/9, 9-5-88, p. 9

[13]  New Scientist 26-3-87, pp 40, 59

[14]  Australasian Health and Healing Vol 7 No 3, April 1988.

[15]  Suggested in Biological Significance of Histocompatibility Antigens, (WHO) Federation Proceedings of the American Association of Biochemists, Vol 31, No 3, 1972, p. 1102.

[16]  Roy Anderson, FRS & Professor Robert May, FRS, New Scientist 26-3-87

[17]  The Central African Journal of Medicine, January 1973, Vol 19 p 1.

[18]  Ibid, p. 4

[19]  Ibid, p. 6

[20]  Ibid, Vol 18, Sept. 1972, pp. 173, 176

[21]  Ibid, July 1972, p. 142

[22]  Sydney Morning Herald, 29-11-1986''
'

         
see also 'AIDS Biowarfare by Dr Alan Cantwell http://sonic.net/~doretk/ArchiveARCHIVE/Aids/1.%20AIDS%20Biowarfare.html

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